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Sagol Department of Neurobiology

Neuroscience research at the Sagol Department of Neurobiology of the University of Haifa is a unique enterprise, focusing on the interface between behavior and its neural substrates.

Each of our research groups, ranging across diverse neuroscience disciplines and model systems, focuses on particular behaviors and seek to unveil their underlying mechanisms. These explorations are conducted on multiple levels, from the molecular and cellular mechanisms to the study of whole neuronal systems.

The methods employed by our faculty include molecular and cellular biology, genetic manipulations, microscopy, in-vitro and in-vivo electrophysiology and human functional imaging. Together, we illuminate topics in learning and memory, and cognitive processes in health and disease from diverse angles, ultimately aiming at understanding the neuronal processes yielding complex behaviors. 

Recent Publications

Santini, E., et al. & Kaphzan, H. (2015). Mitochondrial superoxide contributes to hippocampal synaptic dysfunction and memory deficits in Angelman syndrome model mice. Journal of Neuroscience.

Adaikkan, C., & Rosenblum, K. (2015). A molecular mechanism underlying gustatory memory trace for an association in the insluar cortex. eLife.

Ritov, G., Boltyansky, B., & Richter-Levin, G. (2015). Anovel approach to PTSD modeling in rats reveals alternating patterns of limbic activity in different types of stress reaction. Molecular Psychiatry.   

Maroun, M., & Wagner S. (2015). Oxytocin and memory of emotional stimuli: some dance to remember, some dance to forget. Biological Psychiatry.

Tendler, A., & Wagner, S. (2015). Different types of theta rhythmicity are induced by social and fearful stimuli in a network associated with social memory. eLife.

Awad W. Ferreira, G. & Maroun M. (2015). Dissociation of the Role of Infralimbic Cortex in Learning and Consolidation of Extinction of Recent and Remote Aversion Memory. Neuropsychopharmacology, 15. 

Ghosh S. Reuveni I. Lamprecht R. & Barkai E. (2015). Persistent CaMKII activation mediates learning-induced long-lasting enhancement of synaptic inhibition. Journal of Neuroscience, 35: 128:139.

Ganea DA. Dines M. Basu S. &  Lamprecht R. (2015). The Membrane Proximal Region of AMPA Receptors in Lateral Amygdala is Essential for Fear Memory Formation. Neuropsychopharmacplogy, 15.